No expression of B7-2 was detected in intercapillary cells, vascular endothelial cells and nephric tubule endothelial.

系膜细胞，肾小管上皮细胞和血管内皮细胞表达均阴性。

Hypercalciuria was considered as a secondary condition when (It was) associated with familial renal tubular acidosis.

高钙尿如伴有家族性肾小管性酸中毒，就可以认为是继发病。

The effect of rhein in lipidemia and mitochondrial of renal tubular epithelia was more strong than rosiglitazone.

大黄酸对脂代谢紊乱及肾小管线粒体的格列酮。

Objective To study the cytopathogenic effect of epidemic hemorrhagic fever with renal syndrome virus (HFRSV) on renal tubular cells(RTC).

目的研究肾综合征出血热病毒（HFRSV）对肾小管上皮细胞（RTC）的致病。

These results suggest that early neonatal hopocalcemia appears to be related to a transient lack of response to PTH of the target organs, disordered vitamin D metabolism and hypercalcitoninemia.

由上述结果推论新生儿之血钙调节是由多种因素共同参与，而导致新生儿低血钙症之重要因素，包括暂时性肾小管细胞对副甲状腺素之低反应及维生素D代谢上之障碍。

No expression of B7-2 was detected in intercapillary cells, vascular endothelial cells and nephric tubule endothelial.

系膜细胞，肾小管上皮细胞和血管内皮细胞表达均阴性。

Hypercalciuria was considered as a secondary condition when (It was) associated with familial renal tubular acidosis.

高钙尿如伴有家族性肾小管性中，就可以认为是继发。

The effect of rhein in lipidemia and mitochondrial of renal tubular epithelia was more strong than rosiglitazone.

脂代谢紊乱及肾小管线粒体的作用明优于罗格列酮。

Objective To study the cytopathogenic effect of epidemic hemorrhagic fever with renal syndrome virus (HFRSV) on renal tubular cells(RTC).

目的研究肾综合征出血热（HFRSV）肾小管上皮细胞（RTC）的致作用。

These results suggest that early neonatal hopocalcemia appears to be related to a transient lack of response to PTH of the target organs, disordered vitamin D metabolism and hypercalcitoninemia.

由上述结果推论新生儿之血钙调节是由多种因素共同参与，而导致新生儿低血钙症之重要因素，包括暂时性肾小管细胞副甲状腺素之低反应及维生素D代谢上之障碍。

No expression of B7-2 was detected in intercapillary cells, vascular endothelial cells and nephric tubule endothelial.

系膜细胞，小管上皮细胞和血管内皮细胞表达均阴性。

Hypercalciuria was considered as a secondary condition when (It was) associated with familial renal tubular acidosis.

高钙尿如伴有家族性小管性酸中毒，就可以认为是继发病。

The effect of rhein in lipidemia and mitochondrial of renal tubular epithelia was more strong than rosiglitazone.

大黄酸对脂代谢紊小管线粒体的作用明优于罗格列酮。

Objective To study the cytopathogenic effect of epidemic hemorrhagic fever with renal syndrome virus (HFRSV) on renal tubular cells(RTC).

目的研合征出血热病毒（HFRSV）对小管上皮细胞（RTC）的致病作用。

These results suggest that early neonatal hopocalcemia appears to be related to a transient lack of response to PTH of the target organs, disordered vitamin D metabolism and hypercalcitoninemia.

由上述结果推论新生儿之血钙调节是由多种因素共同参与，而导致新生儿低血钙症之重要因素，包括暂时性小管细胞对副甲状腺素之低反应维生素D代谢上之障碍。

No expression of B7-2 was detected in intercapillary cells, vascular endothelial cells and nephric tubule endothelial.

系膜细胞，肾小管上皮细胞和血管内皮细胞表达均阴性。

Hypercalciuria was considered as a secondary condition when (It was) associated with familial renal tubular acidosis.

高钙尿如伴有家族性肾小管性中，就可以认为是继发。

The effect of rhein in lipidemia and mitochondrial of renal tubular epithelia was more strong than rosiglitazone.

大脂代谢紊乱及肾小管线粒体的作用明优于罗格列酮。

Objective To study the cytopathogenic effect of epidemic hemorrhagic fever with renal syndrome virus (HFRSV) on renal tubular cells(RTC).

目的研究肾综合征出血（HFRSV）肾小管上皮细胞（RTC）的致作用。

These results suggest that early neonatal hopocalcemia appears to be related to a transient lack of response to PTH of the target organs, disordered vitamin D metabolism and hypercalcitoninemia.

由上述结果推论新生儿之血钙调节是由多种因素共同参与，而导致新生儿低血钙症之重要因素，包括暂时性肾小管细胞副甲状腺素之低反应及维生素D代谢上之障碍。

No expression of B7-2 was detected in intercapillary cells, vascular endothelial cells and nephric tubule endothelial.

系膜细胞，管上皮细胞和血管内皮细胞表达均阴性。

Hypercalciuria was considered as a secondary condition when (It was) associated with familial renal tubular acidosis.

高钙尿如伴有家族性管性酸中毒，就可以认为是继发病。

The effect of rhein in lipidemia and mitochondrial of renal tubular epithelia was more strong than rosiglitazone.

大黄酸对脂代谢紊乱管线粒体的作用明优于罗格列酮。

Objective To study the cytopathogenic effect of epidemic hemorrhagic fever with renal syndrome virus (HFRSV) on renal tubular cells(RTC).

目的综合征出血热病毒（HFRSV）对管上皮细胞（RTC）的致病作用。

These results suggest that early neonatal hopocalcemia appears to be related to a transient lack of response to PTH of the target organs, disordered vitamin D metabolism and hypercalcitoninemia.

由上述结果推论新生儿之血钙调节是由多种因素共同参与，而导致新生儿低血钙症之重要因素，包括暂时性管细胞对副甲状腺素之低反应维生素D代谢上之障碍。

No expression of B7-2 was detected in intercapillary cells, vascular endothelial cells and nephric tubule endothelial.

系膜细胞，肾小管上皮细胞和血管内皮细胞表达均阴。

Hypercalciuria was considered as a secondary condition when (It was) associated with familial renal tubular acidosis.

高钙尿如伴有家族肾小管毒，就可以认为是继发病。

The effect of rhein in lipidemia and mitochondrial of renal tubular epithelia was more strong than rosiglitazone.

大黄对脂代谢紊乱及肾小管线粒体的明优于罗格列酮。

Objective To study the cytopathogenic effect of epidemic hemorrhagic fever with renal syndrome virus (HFRSV) on renal tubular cells(RTC).

目的研究肾综合征出血热病毒（HFRSV）对肾小管上皮细胞（RTC）的致病。

These results suggest that early neonatal hopocalcemia appears to be related to a transient lack of response to PTH of the target organs, disordered vitamin D metabolism and hypercalcitoninemia.

上述结果推论新生儿之血钙调节是多种因素共同参与，而导致新生儿低血钙症之重要因素，包括暂时肾小管细胞对副甲状腺素之低反应及维生素D代谢上之障碍。

No expression of B7-2 was detected in intercapillary cells, vascular endothelial cells and nephric tubule endothelial.

系膜，肾小管上和血管内表达均阴性。

Hypercalciuria was considered as a secondary condition when (It was) associated with familial renal tubular acidosis.

高钙尿如伴有家族性肾小管性酸中毒，就可以认为是继发病。

The effect of rhein in lipidemia and mitochondrial of renal tubular epithelia was more strong than rosiglitazone.

大黄酸对脂代谢紊乱及肾小管线粒体的作用明优于罗格列酮。

Objective To study the cytopathogenic effect of epidemic hemorrhagic fever with renal syndrome virus (HFRSV) on renal tubular cells(RTC).

目的研究肾综合征出血热病毒（HFRSV）对肾小管上（RTC）的致病作用。

These results suggest that early neonatal hopocalcemia appears to be related to a transient lack of response to PTH of the target organs, disordered vitamin D metabolism and hypercalcitoninemia.

由上述结果推论新生儿之血钙调节是由多共同参与，而导致新生儿低血钙症之重要，包括暂时性肾小管对副甲状腺之低反应及维生D代谢上之障碍。

No expression of B7-2 was detected in intercapillary cells, vascular endothelial cells and nephric tubule endothelial.

系膜细胞，肾小管上皮细胞和血管内皮细胞表达均阴性。

Hypercalciuria was considered as a secondary condition when (It was) associated with familial renal tubular acidosis.

高钙尿如伴有家族性肾小管性酸中毒，就可以认为是继发病。

The effect of rhein in lipidemia and mitochondrial of renal tubular epithelia was more strong than rosiglitazone.

大黄酸对脂代谢紊乱及肾小管线粒体明优列酮。

Objective To study the cytopathogenic effect of epidemic hemorrhagic fever with renal syndrome virus (HFRSV) on renal tubular cells(RTC).

目研究肾综合征出血热病毒（HFRSV）对肾小管上皮细胞（RTC）致病。

These results suggest that early neonatal hopocalcemia appears to be related to a transient lack of response to PTH of the target organs, disordered vitamin D metabolism and hypercalcitoninemia.

由上述结果推论新生儿之血钙调节是由多种因素共同参与，而导致新生儿低血钙症之重要因素，包括暂时性肾小管细胞对副甲状腺素之低反应及维生素D代谢上之障碍。

No expression of B7-2 was detected in intercapillary cells, vascular endothelial cells and nephric tubule endothelial.

系膜细胞，肾小管上皮细胞和血管内皮细胞表达均阴性。

Hypercalciuria was considered as a secondary condition when (It was) associated with familial renal tubular acidosis.

高钙尿如伴有家族性肾小管性酸中，以认为是继发。

The effect of rhein in lipidemia and mitochondrial of renal tubular epithelia was more strong than rosiglitazone.

大黄酸对脂代谢紊乱及肾小管线粒体作用明优于罗格列酮。

Objective To study the cytopathogenic effect of epidemic hemorrhagic fever with renal syndrome virus (HFRSV) on renal tubular cells(RTC).

目研究肾综合征出血热（HFRSV）对肾小管上皮细胞（RTC）作用。

These results suggest that early neonatal hopocalcemia appears to be related to a transient lack of response to PTH of the target organs, disordered vitamin D metabolism and hypercalcitoninemia.

由上述结果推论新生儿之血钙调节是由多种因素共同参与，而导新生儿低血钙症之重要因素，包括暂时性肾小管细胞对副甲状腺素之低反应及维生素D代谢上之障碍。