A parathormone (PTH) assay reveals a high normal to elevated level of PTH.

甲状旁腺激素(PTH)测定显示PTH升高。

Tetany, epilepsy, neuro-psychiatrical symptoms, hypocalcemia, hyperphosphatemia were common clinical manifestation.There was also low level of PTH, except pseudohypoparathyroidism.

临床表现为手足抽搐、癫痫发作、精神症状、血钙降低、血磷升高、除假性甲旁减外血PTH均降低。

These results suggest that early neonatal hopocalcemia appears to be related to a transient lack of response to PTH of the target organs, disordered vitamin D metabolism and hypercalcitoninemia.

由生儿之血钙调节是由多种因素共同参与，而导致生儿低血钙症之重要因素，包括暂时性肾小管细胞对副甲状腺素之低反应及维生素D代谢之障碍。

A parathormone (PTH) assay reveals a high normal to elevated level of PTH.

甲状旁腺激素(PTH)测定显示PTH升高。

Tetany, epilepsy, neuro-psychiatrical symptoms, hypocalcemia, hyperphosphatemia were common clinical manifestation.There was also low level of PTH, except pseudohypoparathyroidism.

临床表现为手足抽搐、癫痫发作、精神症状、血钙降低、血磷升高、除假性甲旁减外血PTH均降低。

These results suggest that early neonatal hopocalcemia appears to be related to a transient lack of response to PTH of the target organs, disordered vitamin D metabolism and hypercalcitoninemia.

由上述结推论新生儿之血钙调节是由多种因素共同参与，而导致新生儿低血钙症之重要因素，包括暂时性肾小管细胞对副甲状腺素之低反应及维生素D代谢上之障碍。

A parathormone (PTH) assay reveals a high normal to elevated level of PTH.

甲状旁腺激素(PTH)测定显示PTH升高。

Tetany, epilepsy, neuro-psychiatrical symptoms, hypocalcemia, hyperphosphatemia were common clinical manifestation.There was also low level of PTH, except pseudohypoparathyroidism.

临床表现为手足、痫发作、精神症状、血钙降低、血磷升高、除假性甲旁减外血PTH均降低。

These results suggest that early neonatal hopocalcemia appears to be related to a transient lack of response to PTH of the target organs, disordered vitamin D metabolism and hypercalcitoninemia.

由上述结推论新生儿血钙调节是由多种因素共同参与，而导致新生儿低血钙症因素，包括暂时性肾小管细胞对副甲状腺素低反应及维生素D代谢上障碍。

A parathormone (PTH) assay reveals a high normal to elevated level of PTH.

旁(PTH)测定显示PTH升高。

Tetany, epilepsy, neuro-psychiatrical symptoms, hypocalcemia, hyperphosphatemia were common clinical manifestation.There was also low level of PTH, except pseudohypoparathyroidism.

临床表现为手足抽搐、癫痫发作、精神症、血钙降低、血磷升高、除假性旁减外血PTH均降低。

These results suggest that early neonatal hopocalcemia appears to be related to a transient lack of response to PTH of the target organs, disordered vitamin D metabolism and hypercalcitoninemia.

由上述结推论新生儿之血钙调节是由多种因共同参与，而导致新生儿低血钙症之重要因，包括暂时性肾小管细胞对之低反应及维生D代谢上之障碍。

A parathormone (PTH) assay reveals a high normal to elevated level of PTH.

甲状旁腺激素(PTH)测定显示PTH升高。

Tetany, epilepsy, neuro-psychiatrical symptoms, hypocalcemia, hyperphosphatemia were common clinical manifestation.There was also low level of PTH, except pseudohypoparathyroidism.

临床表现为手足、痫发作、精神症状、血钙降低、血磷升高、除假性甲旁减外血PTH均降低。

These results suggest that early neonatal hopocalcemia appears to be related to a transient lack of response to PTH of the target organs, disordered vitamin D metabolism and hypercalcitoninemia.

由上述结推论新生儿血钙调节是由多种因素共同参与，而导致新生儿低血钙症因素，包括暂时性肾小管细胞对副甲状腺素低反应及维生素D代谢上障碍。

A parathormone (PTH) assay reveals a high normal to elevated level of PTH.

甲状旁腺激(PTH)测定显示PTH升高。

Tetany, epilepsy, neuro-psychiatrical symptoms, hypocalcemia, hyperphosphatemia were common clinical manifestation.There was also low level of PTH, except pseudohypoparathyroidism.

临床表手足抽搐、癫痫发作、精神症状、血钙降低、血磷升高、除假性甲旁减外血PTH均降低。

These results suggest that early neonatal hopocalcemia appears to be related to a transient lack of response to PTH of the target organs, disordered vitamin D metabolism and hypercalcitoninemia.

由上述结推论新生儿之血钙调节是由多种因共同参与，而导致新生儿低血钙症之重要因，暂时性肾小管细胞对副甲状腺之低反应及维生D代谢上之障碍。

A parathormone (PTH) assay reveals a high normal to elevated level of PTH.

甲状旁腺激素(PTH)测定显示PTH升高。

Tetany, epilepsy, neuro-psychiatrical symptoms, hypocalcemia, hyperphosphatemia were common clinical manifestation.There was also low level of PTH, except pseudohypoparathyroidism.

临床表现为手足抽搐、癫痫发作、精神症状、钙降、升高、除假性甲旁减外PTH均降。

These results suggest that early neonatal hopocalcemia appears to be related to a transient lack of response to PTH of the target organs, disordered vitamin D metabolism and hypercalcitoninemia.

由上述结推论新生儿之钙调节是由多种因素共，而导致新生儿钙症之重要因素，包括暂时性肾小管细胞对副甲状腺素之反应及维生素D代谢上之障碍。

A parathormone (PTH) assay reveals a high normal to elevated level of PTH.

甲状旁腺激素(PTH)测定显示PTH升高。

Tetany, epilepsy, neuro-psychiatrical symptoms, hypocalcemia, hyperphosphatemia were common clinical manifestation.There was also low level of PTH, except pseudohypoparathyroidism.

临床表现为手足抽搐、癫痫发作、精神症状、血钙降低、血磷升高、甲旁减外血PTH均降低。

These results suggest that early neonatal hopocalcemia appears to be related to a transient lack of response to PTH of the target organs, disordered vitamin D metabolism and hypercalcitoninemia.

由上述结推论新生儿之血钙调节是由素共同参与，而导致新生儿低血钙症之重要素，包括暂时肾小管细胞对副甲状腺素之低反应及维生素D代谢上之障碍。

A parathormone (PTH) assay reveals a high normal to elevated level of PTH.

状腺激素(PTH)测定显示PTH升高。

Tetany, epilepsy, neuro-psychiatrical symptoms, hypocalcemia, hyperphosphatemia were common clinical manifestation.There was also low level of PTH, except pseudohypoparathyroidism.

临床表现为手足抽搐、癫痫发作、精神症状、血钙降低、血磷升高、除假性外血PTH均降低。

These results suggest that early neonatal hopocalcemia appears to be related to a transient lack of response to PTH of the target organs, disordered vitamin D metabolism and hypercalcitoninemia.

上述结推论新生儿之血钙调多种因素共同参与，而导致新生儿低血钙症之重要因素，包括暂时性肾小管细胞对副状腺素之低反应及维生素D代谢上之障碍。