Too few proteins lead to night blindness, perifollicular hyperkeratosis, xerophthalmia, keratomolacia, increased morbidity and mortality in young children.

维生素不足可导致夜、周角化过度、异物笥眼炎、儿童发病率我死亡率增高等。

Histopathologic examination showed hyperkeratosis of epidermis, increased thickness of the granular layer and dilation of subpapillary vessels filled with red blood cells.

组织病理学检查示表皮角化过度,呈乳头瘤增生,颗粒层增厚,真皮乳头见大量增生扩张的细血管,管腔内含红细胞,血管周围淋巴细胞浸润。

Too few proteins lead to night blindness, perifollicular hyperkeratosis, xerophthalmia, keratomolacia, increased morbidity and mortality in young children.

维素不足可导致夜盲、毛囊周角化过度、异物笥眼炎、儿童发率我死亡率高等。

Histopathologic examination showed hyperkeratosis of epidermis, increased thickness of the granular layer and dilation of subpapillary vessels filled with red blood cells.

组织检查示表皮角化过度,呈乳头瘤,颗粒层厚,真皮乳头见大扩张的毛细血管,管腔内含红细胞,血管周围淋巴细胞浸润。

Too few proteins lead to night blindness, perifollicular hyperkeratosis, xerophthalmia, keratomolacia, increased morbidity and mortality in young children.

维素不足可导致夜盲、毛囊周角化过度、异物笥眼炎、儿童发病率我死亡率高等。

Histopathologic examination showed hyperkeratosis of epidermis, increased thickness of the granular layer and dilation of subpapillary vessels filled with red blood cells.

组织病理学检查示表皮角化过度,呈乳,颗粒层厚,真皮乳见大量扩张的毛细血管,管腔内含红细胞,血管周围淋巴细胞浸润。

Too few proteins lead to night blindness, perifollicular hyperkeratosis, xerophthalmia, keratomolacia, increased morbidity and mortality in young children.

维生素不足可导致夜盲、毛囊周角化过度、异物笥眼炎、儿童发病我死高等。

Histopathologic examination showed hyperkeratosis of epidermis, increased thickness of the granular layer and dilation of subpapillary vessels filled with red blood cells.

组织病理学检查示表皮角化过度,呈乳头瘤生,颗粒层厚,真皮乳头见大量生扩张的毛,腔内含红胞,周围淋巴胞浸润。

Too few proteins lead to night blindness, perifollicular hyperkeratosis, xerophthalmia, keratomolacia, increased morbidity and mortality in young children.

维生素不足可导致夜盲、毛囊周角化过度、异物笥眼炎、儿童发病率我死亡率增高等。

Histopathologic examination showed hyperkeratosis of epidermis, increased thickness of the granular layer and dilation of subpapillary vessels filled with red blood cells.

组织病理学检查示表皮角化过度,呈乳头瘤增生,颗粒层增厚,真皮乳头见大量增生扩张的毛细血管,管腔内含红细胞,血管周围淋巴细胞浸润。

Too few proteins lead to night blindness, perifollicular hyperkeratosis, xerophthalmia, keratomolacia, increased morbidity and mortality in young children.

维生素不致夜盲、毛囊周角化过度、异物笥眼炎、儿童发病率我死亡率增高等。

Histopathologic examination showed hyperkeratosis of epidermis, increased thickness of the granular layer and dilation of subpapillary vessels filled with red blood cells.

组织病理学检查示表皮角化过度,呈乳头瘤增生,颗粒层增厚,真皮乳头见大量增生扩张的毛细血管,管腔内含红细胞,血管周围淋巴细胞浸润。

Too few proteins lead to night blindness, perifollicular hyperkeratosis, xerophthalmia, keratomolacia, increased morbidity and mortality in young children.

维生素不足可导致夜盲、毛囊周化过度、异物笥眼炎、儿童发病率我死亡率增高等。

Histopathologic examination showed hyperkeratosis of epidermis, increased thickness of the granular layer and dilation of subpapillary vessels filled with red blood cells.

组织病理学检查示化过度,呈头瘤增生,颗粒层增厚,头见大量增生扩张的毛细血管,管腔内含红细胞,血管周围淋巴细胞浸润。

Too few proteins lead to night blindness, perifollicular hyperkeratosis, xerophthalmia, keratomolacia, increased morbidity and mortality in young children.

维生素不足可导致夜盲、毛囊周角化过度、异物笥眼炎、儿童发病率我死亡率增高等。

Histopathologic examination showed hyperkeratosis of epidermis, increased thickness of the granular layer and dilation of subpapillary vessels filled with red blood cells.

组织病理学检查示表皮角化过度,呈乳头瘤增生,颗粒层增厚,真皮乳头见大量增生扩张的毛细血管,管腔内含红细胞,血管周围淋巴细胞浸润。

Too few proteins lead to night blindness, perifollicular hyperkeratosis, xerophthalmia, keratomolacia, increased morbidity and mortality in young children.

维素不足可导致夜盲、毛囊周角化过度、异物笥眼炎、儿童发病率我死亡率增高等。

Histopathologic examination showed hyperkeratosis of epidermis, increased thickness of the granular layer and dilation of subpapillary vessels filled with red blood cells.

组织病理学检查示表皮角化过度,瘤增,层增厚,真皮见大量增扩张的毛细血管,管腔内含红细胞,血管周围淋巴细胞浸润。