No expression of B7-2 was detected in intercapillary cells, vascular endothelial cells and nephric tubule endothelial.

系膜细胞，上皮细胞和血内皮细胞表达均阴性。

Hypercalciuria was considered as a secondary condition when (It was) associated with familial renal tubular acidosis.

高钙尿如伴有家族性性酸中毒，就可以认为是。

The effect of rhein in lipidemia and mitochondrial of renal tubular epithelia was more strong than rosiglitazone.

大黄酸对脂代谢紊乱及线粒体的作用明优于罗格列酮。

Objective To study the cytopathogenic effect of epidemic hemorrhagic fever with renal syndrome virus (HFRSV) on renal tubular cells(RTC).

目的研究综合征出血热毒（HFRSV）对上皮细胞（RTC）的致作用。

These results suggest that early neonatal hopocalcemia appears to be related to a transient lack of response to PTH of the target organs, disordered vitamin D metabolism and hypercalcitoninemia.

由上述结果推论新生儿之血钙调节是由多种因素共同参与，而导致新生儿低血钙症之重要因素，包括暂时性细胞对副甲状腺素之低反应及维生素D代谢上之障碍。

No expression of B7-2 was detected in intercapillary cells, vascular endothelial cells and nephric tubule endothelial.

系膜细胞，肾小管上皮细胞和管内皮细胞表达均阴性。

Hypercalciuria was considered as a secondary condition when (It was) associated with familial renal tubular acidosis.

高钙尿如伴有家族性肾小管性酸中毒，就可以认为是继发病。

The effect of rhein in lipidemia and mitochondrial of renal tubular epithelia was more strong than rosiglitazone.

大黄酸谢紊乱及肾小管线粒体的作用明优于罗格列酮。

Objective To study the cytopathogenic effect of epidemic hemorrhagic fever with renal syndrome virus (HFRSV) on renal tubular cells(RTC).

目的研究肾综合征病毒（HFRSV）肾小管上皮细胞（RTC）的致病作用。

These results suggest that early neonatal hopocalcemia appears to be related to a transient lack of response to PTH of the target organs, disordered vitamin D metabolism and hypercalcitoninemia.

由上述结果推论新生儿之钙调节是由多种因素共同参与，而导致新生儿低钙症之重要因素，包括暂时性肾小管细胞副甲状腺素之低反应及维生素D谢上之障碍。

No expression of B7-2 was detected in intercapillary cells, vascular endothelial cells and nephric tubule endothelial.

系膜，小管上皮和管内皮表达均阴性。

Hypercalciuria was considered as a secondary condition when (It was) associated with familial renal tubular acidosis.

高尿如伴有家族性小管性酸中毒，就可以认为是继发病。

The effect of rhein in lipidemia and mitochondrial of renal tubular epithelia was more strong than rosiglitazone.

大黄酸对脂代谢紊乱及小管线粒体的作用明优于罗格列酮。

Objective To study the cytopathogenic effect of epidemic hemorrhagic fever with renal syndrome virus (HFRSV) on renal tubular cells(RTC).

目的研究综合征出热病毒（HFRSV）对小管上皮（RTC）的致病作用。

These results suggest that early neonatal hopocalcemia appears to be related to a transient lack of response to PTH of the target organs, disordered vitamin D metabolism and hypercalcitoninemia.

由上述结果推论新生儿之调节是由多种因素共同参与，而导致新生儿症之重要因素，包括暂时性小管对副甲状腺素之反应及维生素D代谢上之障碍。

No expression of B7-2 was detected in intercapillary cells, vascular endothelial cells and nephric tubule endothelial.

系膜细胞，肾小管上皮细胞和血管内皮细胞表达均阴性。

Hypercalciuria was considered as a secondary condition when (It was) associated with familial renal tubular acidosis.

高钙尿如伴有家族性肾小管性酸中毒，就可以认为是继发病。

The effect of rhein in lipidemia and mitochondrial of renal tubular epithelia was more strong than rosiglitazone.

大黄酸对脂代谢紊乱及肾小管线粒体的作罗格列酮。

Objective To study the cytopathogenic effect of epidemic hemorrhagic fever with renal syndrome virus (HFRSV) on renal tubular cells(RTC).

目的研究肾综合征出血热病毒（HFRSV）对肾小管上皮细胞（RTC）的致病作。

These results suggest that early neonatal hopocalcemia appears to be related to a transient lack of response to PTH of the target organs, disordered vitamin D metabolism and hypercalcitoninemia.

由上述结果推论新生儿之血钙调节是由多种因素共同参与，而导致新生儿低血钙症之重要因素，包括暂时性肾小管细胞对副甲状腺素之低反应及维生素D代谢上之障碍。

No expression of B7-2 was detected in intercapillary cells, vascular endothelial cells and nephric tubule endothelial.

系膜细胞，肾小管上皮细胞和血管内皮细胞表达均阴。

Hypercalciuria was considered as a secondary condition when (It was) associated with familial renal tubular acidosis.

高钙尿如伴有肾小管酸中毒，就可以认为是继发病。

The effect of rhein in lipidemia and mitochondrial of renal tubular epithelia was more strong than rosiglitazone.

大黄酸对脂代谢紊乱及肾小管线粒体的作用明优于罗格列酮。

Objective To study the cytopathogenic effect of epidemic hemorrhagic fever with renal syndrome virus (HFRSV) on renal tubular cells(RTC).

目的研究肾综合征出血热病毒（HFRSV）对肾小管上皮细胞（RTC）的致病作用。

These results suggest that early neonatal hopocalcemia appears to be related to a transient lack of response to PTH of the target organs, disordered vitamin D metabolism and hypercalcitoninemia.

由上述结新生儿之血钙调节是由多种因素共同参与，而导致新生儿低血钙症之重要因素，包括暂时肾小管细胞对副甲状腺素之低反应及维生素D代谢上之障碍。

No expression of B7-2 was detected in intercapillary cells, vascular endothelial cells and nephric tubule endothelial.

系膜细胞，肾小管上皮细胞和血管内皮细胞表达均阴性。

Hypercalciuria was considered as a secondary condition when (It was) associated with familial renal tubular acidosis.

高钙尿如伴有家族性肾小管性酸中毒，就可以认为是继发病。

The effect of rhein in lipidemia and mitochondrial of renal tubular epithelia was more strong than rosiglitazone.

大黄酸对脂代谢紊乱及肾小管线粒体的作用明优于罗格列酮。

Objective To study the cytopathogenic effect of epidemic hemorrhagic fever with renal syndrome virus (HFRSV) on renal tubular cells(RTC).

目的研究肾综合征出血热病毒（HFRSV）对肾小管上皮细胞（RTC）的致病作用。

These results suggest that early neonatal hopocalcemia appears to be related to a transient lack of response to PTH of the target organs, disordered vitamin D metabolism and hypercalcitoninemia.

由上述结果推论新生儿之血钙调节是由多种因共同参与，而导致新生儿低血钙症之重要因，暂时性肾小管细胞对副甲状腺之低反应及维生D代谢上之障碍。

No expression of B7-2 was detected in intercapillary cells, vascular endothelial cells and nephric tubule endothelial.

系膜细胞，小管上皮细胞和血管内皮细胞表达均阴性。

Hypercalciuria was considered as a secondary condition when (It was) associated with familial renal tubular acidosis.

高钙尿如伴有家族性小管性酸中，就可以认为是继发。

The effect of rhein in lipidemia and mitochondrial of renal tubular epithelia was more strong than rosiglitazone.

酸脂代谢紊乱及小管线粒体的作用明优于罗格列酮。

Objective To study the cytopathogenic effect of epidemic hemorrhagic fever with renal syndrome virus (HFRSV) on renal tubular cells(RTC).

目的研究综合征出血热（HFRSV）小管上皮细胞（RTC）的致作用。

These results suggest that early neonatal hopocalcemia appears to be related to a transient lack of response to PTH of the target organs, disordered vitamin D metabolism and hypercalcitoninemia.

由上述结果推论新生儿之血钙调节是由多种因素共同参与，而导致新生儿低血钙症之重要因素，包括暂时性小管细胞副甲状腺素之低反应及维生素D代谢上之障碍。

No expression of B7-2 was detected in intercapillary cells, vascular endothelial cells and nephric tubule endothelial.

系膜细胞，肾小管上皮细胞和血管内皮细胞表达均阴。

Hypercalciuria was considered as a secondary condition when (It was) associated with familial renal tubular acidosis.

高钙尿如伴有肾小管酸中毒，就可以认为是继发病。

The effect of rhein in lipidemia and mitochondrial of renal tubular epithelia was more strong than rosiglitazone.

大黄酸对脂代谢紊乱及肾小管线粒体的作用明优于罗格列酮。

Objective To study the cytopathogenic effect of epidemic hemorrhagic fever with renal syndrome virus (HFRSV) on renal tubular cells(RTC).

目的研究肾综合征出血热病毒（HFRSV）对肾小管上皮细胞（RTC）的致病作用。

These results suggest that early neonatal hopocalcemia appears to be related to a transient lack of response to PTH of the target organs, disordered vitamin D metabolism and hypercalcitoninemia.

由上述结新生儿之血钙调节是由多种因素共同参与，而导致新生儿低血钙症之重要因素，包括暂时肾小管细胞对副甲状腺素之低反应及维生素D代谢上之障碍。

No expression of B7-2 was detected in intercapillary cells, vascular endothelial cells and nephric tubule endothelial.

系膜细胞，上皮细胞和血内皮细胞表达均阴性。

Hypercalciuria was considered as a secondary condition when (It was) associated with familial renal tubular acidosis.

高钙尿如伴有家族性性酸中毒，就可以认为是继发病。

The effect of rhein in lipidemia and mitochondrial of renal tubular epithelia was more strong than rosiglitazone.

大黄酸对脂代谢紊乱及线粒体的作用明优于罗格列酮。

Objective To study the cytopathogenic effect of epidemic hemorrhagic fever with renal syndrome virus (HFRSV) on renal tubular cells(RTC).

目的研究综合征出血热病毒（HFRSV）对上皮细胞（RTC）的致病作用。

These results suggest that early neonatal hopocalcemia appears to be related to a transient lack of response to PTH of the target organs, disordered vitamin D metabolism and hypercalcitoninemia.

由上述结果推论新之血钙调节是由多种因素共同参与，而导致新血钙症之重要因素，包括暂时性细胞对副甲状腺素之反应及维素D代谢上之障碍。