Some causes of cerebral edema are an large ischemic strokes and meningitis.

脑水肿可能的原因还包括严重脑梗死、脑膜炎。

Another cause is Dressler syndrome which occurs several weeks after a myocardial infarction, or heart attack.

另一个病因是发生于肌梗死数周后的肌梗死后综合征。

Ischemic and infarcted tissue results in the sloughing off of intestinal mucosal tissue, blood, and mucus into the gut.

缺血及梗死使粘膜组织脱落，与血液、粘液一同道。

The displaced part of the brain can compress the anterior cerebral artery, and that can cause an ischemic stroke.

脑移位的部分会压迫脑前动脉，可缺血性脑梗死。

And infratentorial herniations can compress the vital area in the brain stem that controls consciousness, respiration, and heart rate.

幕上脑疝可能压迫脑的供养动脉，造成脑缺血梗死；枕骨孔疝可能压迫脑干的生命中枢，即控制意识、呼吸和率的部位。

STEMI is infarction that extends the entire thickness of the myocardium.

STEMI指肌梗死延伸至肌全层。

Basically, when heart cells die in a myocardial infarction, it leads to massive inflammation that also involves the serous pericardium.

肌梗死肌细胞死亡，引发累及浆膜包的强烈炎症反应。

Your heart is racing or what we call arrhythmias and other cardiac symptoms can present as myocardial infarctions.

你的跳加速，或者我们称之为律不齐，以及其他脏症状可能表现为肌梗死。

It should be noted, however, that this condition is clinically unstable and often precedes a myocardial infarction.

肌肌钙蛋白水平保持正常，或只是轻微升高。然而，应该指出的是，这种疾病在临床上是不稳定的，而且常常发生在肌梗死之前。

In severe cases, blood flow can been completely cut off, which results in myocardial infarction, or the necrosis of a portion of myocardium.

严重的情况下，血流会被完全阻断，引起肌梗死，即肌的部分坏死。

Okay, let's recap - acute pericarditis is usually caused by a idiopathic causes, but can be caused by viruses or after a myocardial infarction.

急性包炎通常原因未明，也可因病毒感染或肌梗死。

In contrast partial occlusion may progress to localized infarction.

相反，部分闭塞可发展为局部梗死。

A prolonged ischemia can lead to death of the heart tissue, known as heart attack or myocardial infarction.

血液供应减少称为缺血。长时间缺血可脏组织死亡，即脏病发作或肌梗死。

However, in a lead looking directly at an acute MI the T waves may flip and become inverted.

然而，直接观察急性肌梗死联，T波可能会翻转并倒置。

This is an extreme case of clockwise rotation and is suggestive of extensive anterior myocardial infarction.

这是顺时针旋转的极端情况，提示广泛的前壁肌梗死。

While ST segment elevation generally resolves within days and T-wave inversion within weeks following the infarction.

而ST段抬高一般在梗死后几天内消失，T波倒置一般在梗死后几周内消失。

Elevation of more than two small squares in the chest leads or one small square in the limb leads, indicates the possibility of myocardial infarction.

胸联＞2个小方格抬高或肢体联＞1个小方格抬高，提示可能肌梗死。

The posterior cerebral artery can also be compressed, which results in ischemic stroke of the occipital part of the brain, which is responsible for processing of visual information.

另外，脑后动脉也会受到压迫，脑枕部缺血性脑梗死；脑枕部区域本来负责视觉信息的处理。

The development of pathological Q waves occurs nine to twelve hours after the onset of occlusion or in the days following infarction.

病理性Q波的发展发生在闭塞开始后9～12小时或梗死后几天。

Late pathological Q waves are a permanent marker of such an MI on the patient's subsequent ECGs.

晚期病理性Q波是患者随后ECG上肌梗死的永久性标志。