Because ELN mutations cause vascular disease but not cognitive abnormalities, these data implicate LIMK1 hemizygosity in imparied visuospatial constructive cognition.

因为ELN突变引起血管，但没有认知异常，这些资料暗示了激酶1半合性基因在视觉空间建构认知障碍中作用。

Right parietal lobe (supramarginal gyrus) was engaged only in the complex task might because this region were involved in visuospatial memory and processing.

顶叶(缘上回)只在复杂任务出现激活，该区可能是视空间记忆和加工重要部位。

Because ELN mutations cause vascular disease but not cognitive abnormalities, these data implicate LIMK1 hemizygosity in imparied visuospatial constructive cognition.

因为ELN突变引起疾病，但没有认知异常，这些资料暗示了激酶1半合基因在视觉空间建构认知障碍中作用。

Right parietal lobe (supramarginal gyrus) was engaged only in the complex task might because this region were involved in visuospatial memory and processing.

顶叶(缘上回)只在复杂任务出现激活，该区可能是视空间记忆和加工重要部位。

Because ELN mutations cause vascular disease but not cognitive abnormalities, these data implicate LIMK1 hemizygosity in imparied visuospatial constructive cognition.

因为ELN突变引起血管的疾病，但没有认知的异常，这些资料暗示了激酶1半合性基因在视觉空认知障碍中的作用。

Right parietal lobe (supramarginal gyrus) was engaged only in the complex task might because this region were involved in visuospatial memory and processing.

顶叶(缘上回)只在复出现激活，该区可能是视空记忆和加工的重要部位。

Because ELN mutations cause vascular disease but not cognitive abnormalities, these data implicate LIMK1 hemizygosity in imparied visuospatial constructive cognition.

ELN变引起血管的疾病，但没有认知的异常，这些资料暗示了激酶1半合性基在视觉空间建构认知障碍中的作用。

Right parietal lobe (supramarginal gyrus) was engaged only in the complex task might because this region were involved in visuospatial memory and processing.

顶叶(缘上回)只在复杂任务出现激活，该区可能是视空间记忆和加工的重要部位。

Because ELN mutations cause vascular disease but not cognitive abnormalities, these data implicate LIMK1 hemizygosity in imparied visuospatial constructive cognition.

因为ELN突变引起血管的疾病，但没有认知的异常，这些资料暗示了激酶1半合性基因在视觉空间建构认知障碍中的作用。

Right parietal lobe (supramarginal gyrus) was engaged only in the complex task might because this region were involved in visuospatial memory and processing.

顶叶(缘上回)只在复杂任务出现激活，该区可能是视空间记忆和加工的重要部位。

Because ELN mutations cause vascular disease but not cognitive abnormalities, these data implicate LIMK1 hemizygosity in imparied visuospatial constructive cognition.

因为ELN突变引起血管的疾病，但没有知的异常，这些资料暗示了激酶1半合性基因在视觉空间知障碍中的作用。

Right parietal lobe (supramarginal gyrus) was engaged only in the complex task might because this region were involved in visuospatial memory and processing.

顶叶(缘上回)只在务出现激活，该区可能是视空间记忆和加工的重要部位。

Because ELN mutations cause vascular disease but not cognitive abnormalities, these data implicate LIMK1 hemizygosity in imparied visuospatial constructive cognition.

因为ELN突变引起血管的疾病，但没有认知的异常，这些示了激酶1半合性基因在视觉空间建构认知障碍中的作用。

Right parietal lobe (supramarginal gyrus) was engaged only in the complex task might because this region were involved in visuospatial memory and processing.

顶叶(缘上回)只在复杂任务出现激活，该区可能是视空间记工的重要部位。

Because ELN mutations cause vascular disease but not cognitive abnormalities, these data implicate LIMK1 hemizygosity in imparied visuospatial constructive cognition.

为ELN突变引起血管的疾病，但没有认知的异常，这些资料暗示了酶1半合性基觉空间建构认知障碍中的作用。

Right parietal lobe (supramarginal gyrus) was engaged only in the complex task might because this region were involved in visuospatial memory and processing.

顶叶(缘上回)只复杂任务出现，区可能是空间记忆和加工的重要部位。

Because ELN mutations cause vascular disease but not cognitive abnormalities, these data implicate LIMK1 hemizygosity in imparied visuospatial constructive cognition.

因为ELN起血管的疾病，但没有认知的异常，这些资料暗示了激酶1半合性基因在视觉空间建构认知障碍中的作用。

Right parietal lobe (supramarginal gyrus) was engaged only in the complex task might because this region were involved in visuospatial memory and processing.

顶叶(缘上回)只在复杂任务出现激活，该区可能是视空间记忆和加工的重要部位。