No expression of B7-2 was detected in intercapillary cells, vascular endothelial cells and nephric tubule endothelial.

细胞，肾小管上皮细胞和血管内皮细胞表达均阴性。

Hypercalciuria was considered as a secondary condition when (It was) associated with familial renal tubular acidosis.

高钙尿如伴有家族性肾小管性酸中毒，就可以认为是继发病。

The effect of rhein in lipidemia and mitochondrial of renal tubular epithelia was more strong than rosiglitazone.

大黄酸对脂代谢紊乱及肾小管线粒体的作用明优于罗格列酮。

Objective To study the cytopathogenic effect of epidemic hemorrhagic fever with renal syndrome virus (HFRSV) on renal tubular cells(RTC).

目的研究肾综合征出血热病毒（HFRSV）对肾小管上皮细胞（RTC）的致病作用。

These results suggest that early neonatal hopocalcemia appears to be related to a transient lack of response to PTH of the target organs, disordered vitamin D metabolism and hypercalcitoninemia.

由上述结果推论新生儿血钙调节是由多种因素共同参与，而导致新生儿低血钙要因素，包括暂时性肾小管细胞对副甲状腺素低反应及维生素D代谢上障碍。

No expression of B7-2 was detected in intercapillary cells, vascular endothelial cells and nephric tubule endothelial.

系膜细胞，肾小细胞和血内细胞表达均阴性。

Hypercalciuria was considered as a secondary condition when (It was) associated with familial renal tubular acidosis.

高钙尿如伴有家族性肾小性酸中毒，就可以认发病。

The effect of rhein in lipidemia and mitochondrial of renal tubular epithelia was more strong than rosiglitazone.

大黄酸对脂代谢紊乱及肾小线粒体的作用明优于罗格列酮。

Objective To study the cytopathogenic effect of epidemic hemorrhagic fever with renal syndrome virus (HFRSV) on renal tubular cells(RTC).

目的研究肾综合征出血热病毒（HFRSV）对肾小细胞（RTC）的致病作用。

These results suggest that early neonatal hopocalcemia appears to be related to a transient lack of response to PTH of the target organs, disordered vitamin D metabolism and hypercalcitoninemia.

由述结果推论新生儿之血钙调节由多种因素共同参与，而导致新生儿低血钙症之重要因素，包括暂时性肾小细胞对副甲状腺素之低反应及维生素D代谢之障碍。

No expression of B7-2 was detected in intercapillary cells, vascular endothelial cells and nephric tubule endothelial.

系膜细胞，肾小管上皮细胞和血管内皮细胞表达均阴性。

Hypercalciuria was considered as a secondary condition when (It was) associated with familial renal tubular acidosis.

高钙尿如伴有家族性肾小管性酸，可以认为是继发。

The effect of rhein in lipidemia and mitochondrial of renal tubular epithelia was more strong than rosiglitazone.

大黄酸对脂代谢紊乱及肾小管线粒体的用明优于罗格列酮。

Objective To study the cytopathogenic effect of epidemic hemorrhagic fever with renal syndrome virus (HFRSV) on renal tubular cells(RTC).

目的研究肾综合征出血热（HFRSV）对肾小管上皮细胞（RTC）的用。

These results suggest that early neonatal hopocalcemia appears to be related to a transient lack of response to PTH of the target organs, disordered vitamin D metabolism and hypercalcitoninemia.

由上述结果推论新生儿之血钙调节是由多种因素共同参与，而导新生儿低血钙症之重要因素，包括暂时性肾小管细胞对副甲状腺素之低反应及维生素D代谢上之障碍。

No expression of B7-2 was detected in intercapillary cells, vascular endothelial cells and nephric tubule endothelial.

系膜细胞，肾小管上皮细胞和血管内皮细胞表达均阴性。

Hypercalciuria was considered as a secondary condition when (It was) associated with familial renal tubular acidosis.

高钙尿有家族性肾小管性酸中毒，就可以认为是继发病。

The effect of rhein in lipidemia and mitochondrial of renal tubular epithelia was more strong than rosiglitazone.

大黄酸对脂代谢紊乱及肾小管线粒体的作用明优于罗格列酮。

Objective To study the cytopathogenic effect of epidemic hemorrhagic fever with renal syndrome virus (HFRSV) on renal tubular cells(RTC).

目的研究肾综合征出血热病毒（HFRSV）对肾小管上皮细胞（RTC）的致病作用。

These results suggest that early neonatal hopocalcemia appears to be related to a transient lack of response to PTH of the target organs, disordered vitamin D metabolism and hypercalcitoninemia.

由上述结果推论之血钙调节是由多种因素共同参与，而导致低血钙症之重要因素，包括暂时性肾小管细胞对副甲状腺素之低反应及维素D代谢上之障碍。

No expression of B7-2 was detected in intercapillary cells, vascular endothelial cells and nephric tubule endothelial.

系膜细胞，管上皮细胞和血管内皮细胞表达均阴性。

Hypercalciuria was considered as a secondary condition when (It was) associated with familial renal tubular acidosis.

高钙尿如伴有家族性管性酸中毒，就可以认为是继。

The effect of rhein in lipidemia and mitochondrial of renal tubular epithelia was more strong than rosiglitazone.

黄酸脂代谢紊乱及管线粒体的作用明优于罗格列酮。

Objective To study the cytopathogenic effect of epidemic hemorrhagic fever with renal syndrome virus (HFRSV) on renal tubular cells(RTC).

目的研究综合征出血热毒（HFRSV）管上皮细胞（RTC）的致作用。

These results suggest that early neonatal hopocalcemia appears to be related to a transient lack of response to PTH of the target organs, disordered vitamin D metabolism and hypercalcitoninemia.

由上述结果推论新生儿之血钙调节是由多种因素共同参与，而导致新生儿低血钙症之重要因素，包括暂时性管细胞副甲状腺素之低反应及维生素D代谢上之障碍。

No expression of B7-2 was detected in intercapillary cells, vascular endothelial cells and nephric tubule endothelial.

系膜细胞，肾小上细胞和血细胞表达均阴性。

Hypercalciuria was considered as a secondary condition when (It was) associated with familial renal tubular acidosis.

高钙尿如伴有家族性肾小性酸中毒，就可以认为是继发病。

The effect of rhein in lipidemia and mitochondrial of renal tubular epithelia was more strong than rosiglitazone.

大黄酸对脂代谢紊乱及肾小线粒体的作用明优于罗格列酮。

Objective To study the cytopathogenic effect of epidemic hemorrhagic fever with renal syndrome virus (HFRSV) on renal tubular cells(RTC).

目的研究肾综合征出血热病毒（HFRSV）对肾小上细胞（RTC）的致病作用。

These results suggest that early neonatal hopocalcemia appears to be related to a transient lack of response to PTH of the target organs, disordered vitamin D metabolism and hypercalcitoninemia.

由上述结果推论新生儿之血钙调节是由多种因参与，而导致新生儿低血钙症之重要因，包括暂时性肾小细胞对副甲状腺之低反应及维生D代谢上之障碍。

No expression of B7-2 was detected in intercapillary cells, vascular endothelial cells and nephric tubule endothelial.

膜细胞，肾小管上皮细胞和血管内皮细胞表达均阴性。

Hypercalciuria was considered as a secondary condition when (It was) associated with familial renal tubular acidosis.

高钙尿如伴有家族性肾小管性酸中毒，就可以认为是继发病。

The effect of rhein in lipidemia and mitochondrial of renal tubular epithelia was more strong than rosiglitazone.

大黄酸对脂代谢紊乱及肾小管线粒体的作用明优于罗格列酮。

Objective To study the cytopathogenic effect of epidemic hemorrhagic fever with renal syndrome virus (HFRSV) on renal tubular cells(RTC).

目的研究肾综合征出血热病毒（HFRSV）对肾小管上皮细胞（RTC）的致病作用。

These results suggest that early neonatal hopocalcemia appears to be related to a transient lack of response to PTH of the target organs, disordered vitamin D metabolism and hypercalcitoninemia.

由上述结果推论新生儿血钙调节是由多种因素共同参与，而导致新生儿低血钙症因素，包括暂时性肾小管细胞对副甲状腺素低反应及维生素D代谢上障碍。

No expression of B7-2 was detected in intercapillary cells, vascular endothelial cells and nephric tubule endothelial.

系膜细胞，肾小管上皮细胞和血管内皮细胞表达均阴性。

Hypercalciuria was considered as a secondary condition when (It was) associated with familial renal tubular acidosis.

高钙尿如伴有家族性肾小管性酸中毒，就可以认为是继发病。

The effect of rhein in lipidemia and mitochondrial of renal tubular epithelia was more strong than rosiglitazone.

大黄酸对脂代谢紊乱及肾小管线粒体的作用明优于罗格列酮。

Objective To study the cytopathogenic effect of epidemic hemorrhagic fever with renal syndrome virus (HFRSV) on renal tubular cells(RTC).

目的研究肾综合征出血热病毒（HFRSV）对肾小管上皮细胞（RTC）的致病作用。

These results suggest that early neonatal hopocalcemia appears to be related to a transient lack of response to PTH of the target organs, disordered vitamin D metabolism and hypercalcitoninemia.

由上述结果推论新生儿之血钙调节是由多种素共同参与，而导致新生儿低血钙症之素，包括暂时性肾小管细胞对副甲状腺素之低反应及维生素D代谢上之障碍。

No expression of B7-2 was detected in intercapillary cells, vascular endothelial cells and nephric tubule endothelial.

系膜，肾小管上皮和血管内皮表达均阴性。

Hypercalciuria was considered as a secondary condition when (It was) associated with familial renal tubular acidosis.

高钙尿如伴有家族性肾小管性酸中毒，就为是继发病。

The effect of rhein in lipidemia and mitochondrial of renal tubular epithelia was more strong than rosiglitazone.

大黄酸对脂代谢紊乱及肾小管线粒体作用明优于罗格列酮。

Objective To study the cytopathogenic effect of epidemic hemorrhagic fever with renal syndrome virus (HFRSV) on renal tubular cells(RTC).

目研究肾综合征出血热病毒（HFRSV）对肾小管上皮（RTC）致病作用。

These results suggest that early neonatal hopocalcemia appears to be related to a transient lack of response to PTH of the target organs, disordered vitamin D metabolism and hypercalcitoninemia.

由上述结果推论新生儿之血钙调节是由多种因素共同参与，而导致新生儿低血钙症之重要因素，包括暂时性肾小管对副甲状腺素之低反应及维生素D代谢上之障碍。